In our HF treatment, IPA analysis suggested PPARGC1A regulation of Carnitine Palmitoyltransferase 1B (CPT1B), glycerol kinase (GK), and cluster of differentiation 36 (CD36), all DEGs related to FA uptake, triglyceride metabolism, and fatty acid oxidation (FAO) [71–74]. The gene discussed is CD36; the disease is hydrops fetalis.