CPT1A and metabolic dysfunction-associated steatotic liver disease: Furthermore, in the context of NAFLD, lentiviral-mediated TAF9 overexpression changed the protein and mRNA levels of mediators of fatty acid β-oxidation and LD accumulation, as evidenced by increased CPT1A, ACOX1 and PPARα expression and decreased ACSL1 and ADRP expression (Figures 5H–K).