Whilst there is a slight advantage to the AF treatment in the number of genes associated with the subset of postulated progenitor‐related p63‐associated genes38 (50 in AF vs. 23 in FCS, besides 67 shared), it is not clear that the two treatments result in a difference of the categorical distribution of keratinocytes along the proliferation differentiation axis. This evidence concerns the gene TP63 and atrial fibrillation.