Changes in various lipid levels were found in subjects with non–small cell lung cancer (NSCLC) after tumor resection, further suggested role of lipid metabolism in tumor progression [68] and pointing it as a treatment target [69].Additionally, we observed an increase in the catabolic pathway of BCAA in HMP cell lines manifested by the accumulation of their products of catabolism in both cell and growth media. This evidence concerns the gene ARID4B and non-small cell lung carcinoma.