MTOR and neoplasm: Using CDH3 expression as continuous label, we found that CDH3‐positively correlated genes were enriched for signatures of genes upregulated upon the activation of the WNT‐β‐catenin signaling, RTK/PI3K/mTOR signaling axis (through ERBB2, VEGF and mTOR), and upon inactivation of the tumor‐suppressor genes PTEN and RB; in addition, CDH3‐positively correlated genes were enriched for signatures of genes downregulated upon HOXA9 silencing (Fig. 4A).