We found a nearly perfect antisense complementarity for the entire CoV2‐miR‐O7a.2 sequence to the 3′ untranslated regions (3′UTR) of BATF2 with 100% complementarity in the seed region (Fig 4C), which is a transcription factor that plays a major role in innate immunity during viral infection (Tussiwand et al, 2012; Murphy et al, 2013). The gene discussed is BATF2; the disease is viral infectious disease.