Those findings in inflammatory biomarkers, oxygen saturation, and pulmonary artery systolic pressure in our study and previous studies (27, 28, 31, 32) support the observations in the present study that no significant differences exist in cTnI concentration, and echocardiographic structural and functional parameters among COVID-19 survivors, healthy control, and risk factor-matched control 327 days after diagnosis of COVID-19 infection. The gene discussed is TNNI3; the disease is COVID-19.