STIM1 and cardiomyopathy: Among those single molecules, we identified the stromal interaction molecule 1 (STIM1) precursor, which increased in abundance by 5.4-fold among patients with SARS-CoV-2 infection compared with patients with noninflammatory cardiomyopathy (log2 fold change, 2.43; −log10 [P value] = 10.05) (eFigure 3A in Supplement 1).