In breast cancer stem‐like cells, METTL14 mRNA stability and protein expression are upregulated by overexpression of the upstream molecule AURKA, which stabilizes METTL14 protein by inhibiting ubiquitylation‐dependent degradation of METTL14, resulting in enhanced DROSHA mRNA stability through METTL14‐dependent m6A modification. The gene discussed is METTL14; the disease is breast carcinoma.