Our data suggest that ACE2 is already expressed in MCs in basal conditions, and the modulation of its expression is not a major mechanism regulating SARS-CoV-2 infection in these cells; in fact we found an high expression of ACE2 in uninfected MeT5A cells, and a tendential increase upon infection with SARS-CoV-2. This evidence concerns the gene ACE2 and infection.