TERT and cancer: All TERT mutations were detected in previously described positions and well-known hotspots responsible for the recruitment of transcription factors activating expression of TERT in cancer, including 2 double substitutions c.-139_-138delinsAA, 9 substitutions c.-146G>A, 2 substitutions c.-101G>A, and 3 other substitutions (c.-150G>A, c.-100G>A, and c.-99G>A).