A cross-sectional study measuring 1H-MRS recruited 188 outpatients and indicated that liver triglyceride content and IR were the main reasons for ApoB elevation; worse atherogenic dyslipidemia was predicted by the degree of liver fat accumulation, adipose tissue, and systemic IR, but severe steatohepatitis did not lead to a worse atherogenic lipid profile (17). This evidence concerns the gene APOB and metabolic syndrome.