The strong decrease in the hepatic expression of PCTP could be the cause of a possible degradation of glucose homeostasis in fetuses of H recipients (CH and HH), since the increase in circulating PLTP (phospholipid transfer protein) is described as an inter-organ (brown adipose tissue and liver) mediator to improve glucose tolerance and insulin sensitivity (Sponton et al., 2020). This evidence concerns the gene PCTP and cyclic hematopoiesis.