NEAT1 and Hepatic fibrosis: More importantly, inhibition of NEAT1 upregulated ethanol-induced hepatic function in ASH mice and inhibited lipid, inflammatory responses, hepatocyte apoptosis, and hepatic fibrosis, demonstrating that knockdown of NEAT1 inhibited hepatic fibrosis in ASH mice and thus slowed down the development of ASH (Ye et al., 2020).