On the contrary, overexpression of LINC01093 is involved in the inhibition of nuclear translocation of NF-κB p65 leading to an increase in the nuclear level of NF-κB p65 and a decrease in NF-κB p65 at the cytoplasmic level, and this evidence suggests that overexpression of LINC01093 could be involved in inhibiting hepatocyte apoptosis and attenuating the process of liver fibrosis by suppressing the NF-κB signaling pathway (Shi et al., 2019). Here, LINC01093 is linked to Hepatic fibrosis.