CXCL9 and hereditary sensory and autonomic neuropathy: CXCL9 has been reported to play a pro-neuroinflammation role in the nervous system (Koper et al., 2018), and inhibiting CXCL9 expression could relieve hyperalgesia in BCP rats (Sun et al., 2020), indicating that NONRATT021203.2 could target CXCL9 and result in CIP in the BCP model.