An investigation of the role of FERMT2 breast cancer metastasis showed that it is a direct target of miR-200b; forced expression of miR-200b in a breast cancer cell line expressing high levels of FERMT2 reduced the tumor-forming ability and metastatic properties of the cells when injected into mice, and led to downregulation of several EMT markers [75]. This evidence concerns the gene FERMT2 and breast carcinoma.