Meanwhile, secretion of CCL2, CCL3, CXCL2, and CXCL10 is known to cause large numbers of innate immune cells to enter lung tissue [64], where they release more and more cytokines and amplify the CRS process, thereby damaging the basic lung structure and preventing proper lung function [65,66] (Figure 2). This evidence concerns the gene CCL2 and congenital rubella syndrome.