We have shown previously that this pathogen targets mitochondrial dynamics during infection of primary human monocyte-derived macrophages (hMDMs) by injecting type 4 secretion system (T4SS) effectors such as MitF, leading to a fragmented mitochondrial network via the recruitment of the host fission protein DNM1L to the mitochondrial surface (Escoll et al., 2017b). This evidence concerns the gene MITF and infection.