Studies have shown that carvacrol may induce apoptosis by reducing mitochondrial potential, releasing cytochrome C, activating caspase and carving PARP, thereby inhibiting human metastatic breast cancer cell line (MDA-MB 231) or human non-small cell lung cancer cell line Proliferation (A549) (Suntres et al., 2015). This evidence concerns the gene CYCS and breast carcinoma.