Through analyzing the relationship between risk score and tumor immune infiltration, we further investigated the potential molecular mechanism of risk score in TNBC development, which demonstrated that risk score was positively correlated with macrophage M2, mast cells resting, NK cells activated, mast cells activated, etc., and negatively correlated with T-cell CD4 memory activated, dendritic cells resting, T-cell CD4 memory resting, B-cell naive, etc. (Figure 3(a)). The gene discussed is CD4; the disease is neoplasm.