Loss-of-function mutations of the cystic fibrosis transmembrane conductance regulator (CFTR) anion channel disrupt transepithelial salt-water transport in the lung, intestine, pancreatic duct, and sweat duct, and cause cystic fibrosis (CF), the most common inherited lethal disease among caucasians (O’Sullivan and Freedman, 2009). This evidence concerns the gene CFTR and cystic fibrosis.