The mechanisms of FAK in triggering EMT in GC are proposed to serve as the intersection of multiple signal pathways, including MAPK, phospoinositide 3-kinase (PI3K)/AKT and Wnt/β-catenin (Matsuoka et al., 2012; Dammann et al., 2014; Wang et al., 2019). The gene discussed is AKT1; the disease is gastric cancer.