Recently, we revealed that Epstein-Barr virus latent membrane protein 1 (EBV-LMP1) increases the mitochondrial fission-induced glycolytic metabolic phenotype for NPC cells survival to resist chemotherapy, and phosphorylation of Drp1 Ser616 or dephosphorylation of Drp1Ser637 is essential for LMP1-regulated enhancement of glycolytic metabolism (98). This evidence concerns the gene PDLIM7 and nasopharyngeal carcinoma.