A previous study on a T2DM animal model–the Goto-Kakizaki (GK) rat–demonstrated that 8-week treatment with miglitol, which is an α-glucosidase inhibitor, suppressed postprandial hyperglycemia, in contrast to that in non-treated GK control rats, but it did not improve the insulin secretion capacity and pancreatic exhaustion (43). Here, INS is linked to Hyperglycemia.