Among these potential targets, RELA (RELA proto-oncogene, NF-κB subunit), CASP3, CASP9, BAX, BCL2, CCND1, and CDKNIA were screened as relatively high-level targets (Figure 5), which played an essential role in the inhibition effect of PM on CRC. This evidence concerns the gene CCND1 and colorectal carcinoma.