SOCS3 can bind to specific cytokine receptors and inhibit the JAK/STAT signaling pathway, while RSV virus suppresses the interferon-mediated antiviral response by affecting the protein expression of SOCS1/3, both of which are also considered to be important factors in the anti-RSV infection and acutely exacerbated asthma. Here, SOAT1 is linked to asthma.