Mice data suggested that, during the ALI, the recruiting of CD4+ T lymphocytes to the lung is partly activated by the increased level of IL-16 produced in ECs (lung endothelial cells), whereas the recruiting of neutrophils is inhibited by the increased IL-10 level during these process [33], which supported our observed lower level of NLR in pneumonia children. The gene discussed is IL16; the disease is susceptibility to pneumonia measurement.