The slightly declined TGF-β signaling and MMP-9 expression along with escalated VEGF expression after CB-MNC infusion (Figures 4F,G) implied the prevalence of adopting MNC in both balancing the delicate and intertwining feature of inflammatory networks and rescuing the devastative developing lung in experimental BPD model caused by hyperoxia exposure. The gene discussed is MMP9; the disease is bronchopulmonary dysplasia.