Although some experiments have shown that Verteporfin and VGLL4 mimic peptides that inhibit tumor growth by inhibiting YAP/TAZ transcriptional activity in YAP/TAZ-dependent mouse tumor models, there is also a recent, exciting finding that inhibiting TEADs palmitoylation is likely to block YAP-dependent transcription (105). Here, VGLL4 is linked to neoplasm.