In the 40% monobenzone cream-induced vitiligo model, BA intraperitoneal injection inhibited the infiltration of leukocytes and CD8 + T cells in vitiligo lesions, increased the tyrosinase activity in the lesion area, reduced the expression of chemokine CXCL10 and its receptor CXCR3, and reduced the expression of inflammatory factors in serum samples, including IL-6 and TNF- α, IFN- γ And IL-13 (Zhu et al., 2019). This evidence concerns the gene IL6 and vitiligo.