To assess whether CCL20 stimulation of colorectal cancer cells induces activation of HGF’s receptor, c-Met, we measured expression of c-Met and phosphorylated c-Met by Western blots and found CCL20 to induce c-Met phosphorylation after CCL20 exposure in both a concentration-dependent (Figure 1E) and time-dependent manner (Figure 1F) in both HT-29 and HCT116. This evidence concerns the gene MET and colorectal cancer.