In addition, we found that a novel significantly upregulated circRNA called circ-ATP10A may serve as a promising marker for the prognosis evaluation of patients with MM, and the bioinformatics analyses implied that it may promote MM angiogenesis by sponging hsa-miR-6758-3p/hsa-miR-3977/hsa-miR-6804-3p/hsa-miR-1266-3p/hsa-miR-3620-3p to regulate the expression of VEGFB, HIF1A, PDGFA, and the FGF family. This evidence concerns the gene HIF1A and Miyoshi myopathy.