Taken together with evidence that FGF7 activates FGFR2 in FP‐RMS cells, our data strongly support this ligand‐receptor pairing forming an autocrine loop promoting FP‐RMS cell viability and survival in a similar fashion to that observed in prostate tumors and pre‐adipocytes [56, 57]. The gene discussed is FGFR2; the disease is prostate neoplasm.