These findings may possibly be due to the following reasons: (i) lnc‐UCA1 might regulate Th cell activation in endothelial cells, which resulted in an impaired immune response and thus led to increased inflammation in CHD patients32 (ii) miR‐26a might mediate regulatory T‐cell function and further to suppress immune response under atherosclerotic condition, thus led to reduced inflammatory cytokines in CHD patients.36 Here, UCA1 is linked to coronary artery disorder.