HF was exacerbated in cardiac‐specific CaMKIIdeltaC transgenic mice, inhibition of CaMKII reversed cardiac dysfunction in a genetic model of dilated cardiomyopathy,5, 6, 7, 8, 9 and some strategies which inhibited CaMKII suppressed cardiac hypertrophy,10, 11 and a recent study indicated that chronic CaMKII inhibition with KN‐93, a widely used inhibitor of CaMKII, improved cardiac function and cardiac remodelling in HF mice induced by pressure‐overload.12 Here, CAMK2G is linked to dilated cardiomyopathy.