The ECM‐related pathways included vascular endothelial growth factor (VEGF) signaling, ECM organization, ECM proteoglycans, and TGF‐β signaling, indicating that high Hh activity was associated with TGF‐β‐associated ECM remodeling, a potential promoter for tumor immunosuppression.34 The gene discussed is TGFB1; the disease is neoplasm.