AKT1 and neoplasm: Another example are omental adipocytes (ADI), which promote the homing and invasion of ovarian cancer by multiple mechanisms, including the secretion of adipokines, the promotion of tumour cell metabolism through the direct transfer of lipids to cancer cells9, 10, 11 and the ADI‐induced phosphorylation of salt‐inducible kinase 2 (SIK2), mediating AMPK‐dependent acetyl‐CoA carboxylase phosphorylation and PI3K/AKT activation in tumour cells.12