AlloHSC-iNKT cells without BCAR engineering were able to kill MM.1S-CD1d-FG tumor cells, while PBMC-T cells could not, indicating an intrinsic antitumor NK function of AlloHSC-iNKT cells (Figures 4E, S1D, and S1G); this intrinsic antitumor NK function was inherited by AlloBCAR-iNKT cells, as confirmed by NK activating receptor (i.e., DNAM-1) blocking assay (Figure 4F). The gene discussed is CD226; the disease is neoplasm.