The lead variant at MAU2/TM6SF2 rs73001065 is in linkage disequilibrium (r2 = 0.90) with the missense variant p.E167K at TM6SF2, and the lead variant at PNPLA3 is in high linkage disequilibrium (r2 = 0.98) with the missense variant p.I148M at PNPLA3. Table 1 presents the details of these results as well as the effect of other previously associated variants with NAFLD (p.A165T at MTARC1, a splice variant HSD17B13, and another variant at MBOAT7). This evidence concerns the gene MAU2 and metabolic dysfunction-associated steatotic liver disease.