The production of proinflammatory cytokines CXCL10 and IL-6 increased in both HRV16 and HRV1B infection (p < 0.05 for each), whereas IL-8 and IL-18 production increased in HRV1B infection (p = 0.025 and p = 0.018, respectively), but not in HRV16 infection (p = 0.314 and p = 0.674, respectively). Here, IL6 is linked to infection.