In addition, T3s can induce apoptosis in cancer cells through several mechanisms, which includes inhibition of the nuclear factor-κB (NF-κB) pathway and its regulated gene products [10] as well as regulation of both intrinsic and extrinsic apoptotic pathways by modulating the caspase cascades, expression of B cell lymphoma 2 (BCL2), BCL2-associated X protein (BAX), and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) by upregulating the expression of death receptors (DRs) [11,12]. This evidence concerns the gene BAX and cancer.