c-Myc activation appears to be a molecular hallmark of cancer, whereas partial suppression of c-Myc back to its physiological levels can result in sustained tumor regression and is associated with tumor cells’ proliferative arrest, differentiation, senescence, apoptosis, remodeling of the tumor microenvironment, mediation of immune response, and inhibition of angiogenesis [22,23]. Here, MYC is linked to neoplasm.