NLRP3 and enteritis: When the effect of oral preadministration of L. johnsonii L531 in piglets with Salmonella infantis-induced enteritis was evaluated, S. infantis activated NLRP3 and NF-κB signaling in the jejunum and ileal tissue, L. johnsonii L531 administration before the challenge reduced the severity of intestinal inflammation and prevented the excessive expression of NLRP3 and caspase-1 through the elimination of damaged mitochondria and accelerated autophagic degradation [33].