Chronic stimulation of inflammatory reactions owing to bacterial infection in the gastrointestinal tract generates induction of MetS-related dyslipidemia, triggers release of C-reactive protein (CRP), escalates blood leukocytes and MetS-related homocysteine, increases the concentrations of MetS-related fibrinogen, induces hypercoagulability, stimulates immune cross-reactivity, and rises pro-inflammatory cytokines and other cytotoxic agents. The gene discussed is CRP; the disease is metabolic syndrome.