Previously, our group profiled gene expression in the whole mouse brain after 32 days of infection with T. gondii, and found that expression of CXC chemokine receptor 3 (CXCR3) and its ligand, CXC chemokine ligand 10 (CXCL10), was upregulated by the infection along with other immune response-associated genes [20]. This evidence concerns the gene CXCR3 and infection.