IL-6 is known to have a protective role during early infection with T. gondii and it mediates susceptibility to the parasite [64,65], and IL-12, a well-known proinflammatory cytokine, stimulates IFN-γ synthesis by natural killer cells and T lymphocytes, and plays a critical role in resistance against T. gondii [66,67]. The gene discussed is IL6; the disease is infection.