Several other molecules, such as adiponectin, AICAR, metformin, and resveratrol, were also found to mitigate some pathological features of DMD in cell cultures and animal models, mainly by activating AMPK signaling thus inhibiting NF-κB and reducing inflammasome activation [138,140,141,142,143]. Here, NFKB1 is linked to Duchenne muscular dystrophy.