Notably, in all five tissue types, we fund that the abundance of adoptively transferred eNKs peaked at the same time in both two groups; however, a higher percentage and number of human CD45+CD56+ cells in tumor-bearing mice than in non-tumor-bearing mice (Figure 3b,c) was observed, which suggested that the tumor microenvironment may positively affect proliferation of adoptively transferred eNKs. Here, NCAM1 is linked to neoplasm.