Preliminary studies in mice with derivatives of NeoBOMB1, carrying a clickable residue, have been applied in combination with the radiolabeled complementary small molecule [111In]In-Tz, and thus far, have failed to improve tumor-to-pancreas ratios, most likely due to undesired folding of the targeting vector in the binding pocket of the GRPR [113]. This evidence concerns the gene GRPR and neoplasm.