FSCN1 is regulated by several signaling pathways (AMPK/mTOR, Wnt/β-catenin, and MAPK) and is overexpressed in various human carcinomas including gynecological cancers; however, understanding the exact molecular mechanisms underlying FSCN1 deregulation and interaction with other genes and oncoviruses, especially in gynecological cancers, is still nascent. This evidence concerns the gene MTOR and female reproductive organ cancer.