BCP significantly increased caspase-3 and Bax, whereas it reduced Bcl-2 expression, compared with untreated cells in both MM.1S and MM.1R following 24 h of treatment especially at a concentration of 100 μM, thus indicating that BCP induced the apoptotic process in MM cancer cells (p < 0.05 vs. CTRL; Figure 4). The gene discussed is BCL2; the disease is cancer.